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This is achieved by the ability of erythrocytes to scavenge and detoxify exogenous neutrophils-, monocytes- or endothelial cells-derived reactive oxygen species (ROS) and reactive nitrogen species (RNS) [53, 54]

One possible explanation for such a response is that inhibition of mTORC1 and activation of GCN2 collectively suppresses global translation to conserve nutrients, while selective translation of mRNA such as ATF4 (mediated by increased p-eIF2) activates the expression of AARE genes to mitigate cellular stress

10.3389/fcell.2024.1372330 53 SunK.LiC.LiaoS.YaoX.OuyangY.LiuY.et al (2022)

[Google Scholar] 22.Kalman DS, Incledon T, Gaunaurd I

Exp Cell Res 2010

Factor XIIIa inhibitors Numerous synthetic and natural factor XIII (FXIII) inhibitors have been investigated in preclinical trials and in vitro studies