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doi:10.1016/j.cancergencyto.2004.04.008 Antonescu CR, Nafa K, Segal NH, Dal Cin P, Ladanyi M

This effect seems to be exacerbated in the context of microbial dysbiosis

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Depletion of GSH levels necessary for adequate protection to less than 20-30% has been suggested as an explanation of hepatotoxicity of higher doses of compounds such as paracetamol or bromobenzene [13]

There are several major enzyme families involved in Phase I metabolism, including: Cytochrome P450 (CYP) enzymes : This is the most prominent family of enzymes involved in Phase I metabolism

The titration protocol exists for safety reasons that are independent of any co-administered compound