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doi:10.1111/dom.15292 Pasternak B, Wintzell V, Hviid A, Eliasson B, Gudbjrnsdottir S, Jonasson C, et al

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DOI: 10.1056/NEJMoa2502082 Cao J, Belousoff MJ, Danev R, Christopoulos A, Wootten D, Sexton PM, et al

CaMKK2 dysfunction may play an underlying role in this phenomenon as genetic deletion of Camkk2 in hippocampal neurons, myeloid cells and immortalized cell lines (HEK293 and HepG2) results in mitochondrial dysfunction that causes a similar switch in cellular energy generation from oxidative phosphorylation to glycolysis [43, 201, 202]

German AJ, Maddison JE, Guilford G

GLP-1 receptor agonists in patients with chronic kidney disease and either overweight or obesity