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When valproate-treated adult rats are examined for PKC phosphorylation of substrates NMDAR1 and MARCKS, there is a significant decrease in immunoblotting tests of phosphorylation by pkc, indicating a downregulation of hippocampal pkc activity due to valproate (Watson et al., 1998)

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Leaky GluR2 in PCs of Lurcher mice leads to increased intracellular Na + [80], which activates the voltage-gated calcium channels and the increased influx of Ca 2+ [31]

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