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Further studies in the 2000s, (7) suggested that the GHK-Cu peptide hosts the potential to not only stimulate the collagen synthesis but also stimulate the production of tissue inhibitors, TIMP-1 and TIMP-2

Their ability to simultaneously address metabolic risk factors, inflammation, and cellular proliferation positions them as one of the most promising drug classes for interrupting SLD progression (Figure 2)

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Two matched the inflammatory eosinophil I and II phenotypes as identified at day 14 p.i., while a third cluster was marked by high expression of pathogen-derived transcripts, suggesting intracellular uptake of parasites (Figs