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Previously, we identified the chemical effects of dielectric barrier discharges (DBD) on the glutathione (GSH) and glutathione disulphide (GSSG) molecules as the most important redox pair in organisms responsible for detoxification of intracellular reactive species

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HATs (histone acetyltransferases) contribute to the regulation of gene expression, and loss or deregulation of these activities may link to tumorigenesis

As OCTN3 mediates sodium-independent carnitine transport and humans lack a functional ortholog of this protein, caution is warranted when extrapolating data on renal carnitine handling from rodent models to humans [78, 79]

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