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These findings provide new insights into the pathogenesis of CM and suggest that microglial GLP-1R in the TNC may be a new target for the treatment of CM

Nevertheless, there is currently a lack of OA-specific genetic models validating the FSP1 or DHODH targets, as well as systematic research on the expression profiles and activity distribution of these pathways in different tissues such as cartilage, synovium, and subchondral bone

Early recognition and communication with healthcare providers ensure any potential risks are addressed promptly, keeping your treatment safe and effective

Therefore, fully understanding the pathogenesis of OA would be helpful in exploring novel therapies

Fatigue often peaks in the first few weeks and improves as your body adjusts

Cagrilintide is a new synthetic peptide analog of the human hormone amylin