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doi: 10.1038/nature08119 97 RuggeriRMVicchioTMCristaniMCertoRCaccamoDAlibrandiAet al

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In hyperthyroidism, accelerated mitochondrial metabolism produces excessive reactive oxygen species (ROS), overwhelming endogenous antioxidant defenses (111, 112)

A splicing mutation has been held responsible for Japanese acatalasemia I where a substitution of a guanine residue with adenine residue at position 5 of intron 4 disturbed the splicing pattern of the RNA product producing a defective protein [137]

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Inflammatory Modulation: Laboratory research indicates BPC-157 may interact with inflammatory signaling pathways in controlled settings