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During oxygenated or oxygen-deprived states, hypoxia-inducible factor 1 (HIF-1) promotes Th17 differentiation by directly transcriptionally activating RORt and facilitating the formation of a tertiary complex comprising RORt, p300, and HIF-1 at the IL-17 promoter region [41]
Retrieved June 28, 2017
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Immune stress accompanied by metabolic remodeling tracks with ASD severity and aligns with evidence of disrupted synaptic function under maladaptive brainimmune coupling 6,7,8
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