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183,184 Both ferroptosis and disulfidptosis depend on intracellular redox homeostasis
UA treatment suppresses the MS inflammatory cascade, the invasion of inflammatory cells into the CNS, and CNS tissue damage in animal models of MS [44]
Studies indicate that FOXO3 mainly regulates manganese superoxide dismutase, whereas catalase expression may depend on FOXO1 or FOXO4
Interestingly, this residue is conserved in Class B1 GPCRs, such as glucagon, vasoactive intestinal peptide (VIP), and glucagon-like peptide-1 (GLP-1) (Liu et al., 2025)
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