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32(2): 3426 Harper P, Elwin CE, Cederblad G

This compound is not approved for human therapeutic use

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Collectively, these observations establish that NRF2 alone can sustain sufficient transcriptional output to maintain cancer cell line growth and that metabolic reprogramming of cells driven by NRF2 does not require redox buffering via enhanced glutathione production

NINDS Program staff has indicated that proposals submitted in September 2025 may be selected and funded before the end of Fiscal Year 2026

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