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study found that 11.9% of patients experienced worsening, 0.4% saw resolution, and 0.8% stabilized during the study period, the relatively short follow-up means many participants were still in the active shedding phase when assessed

However, bearing in mind that the gliA promoter lacks of an obvious GliZ binding motif, the correlation between gliZ expression and gliotoxin biosynthesis, the essential role of ZafA for gliA expression during zinc starvation and the close proximity of the ZafA and GipA binding sites in the gliA promoter, it would appear more feasible that GipA, whose activity could also be influenced by the intracellular amount of gliotoxin, plays a role as a modulator of the ZafA-mediated induction of gliA under zinc starvation to adjust the gliA expression level to the intracellular amount of gliotoxin

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