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Toxic lipid peroxidation products and oxidative stress caused by peroxides are problems for yeast strains that are either GSH-deficient or have changes to their GSH redox status

Today 3 , 206214 (1987)

The extent of PDI degradation in response to mild levels of oxidative stress was 90% greater over the span of 24 h than that of other proteins

Endothelial cell dysfunction and the pathobiology of atherosclerosis

This observation was mechanistically determined to be in part due to A 142 binding directly to COX1 and inducing loss of enzymatic activity of the greater mitochondrial electron transport chain complex 4 (C4), of which COX1 is an integral part

10.1172/JCI99490 31 de CarvalhoC.CaramujoM