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However, since CD38 could be expressed by other immune and non-immune cells 60,61 , our results suggest that the efficacy of targeting CD38 might not solely rely on CD38 expression level in T cells, opening up additional therapeutic opportunities

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Importantly, by immunohistochemical (IHC) staining of xenograft tumor tissues, we found that knocking down ACTL6A expression significantly decreased the level of Ki67, a proliferation marker, and increased that of 4-hydroxy-2-noneal (4-HNE) 45 , a lipid peroxidation marker (Fig