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doi:10.1111/j.1365-2958.2009.06955.x 47
(100) subjected PS1 gene-knockin mice to continuous EODF for 3 months and found that the EODF group exhibited enhanced resistance to excitotoxic damage in hippocampal CA1 and CA3 neurons, with fewer lipid peroxidation products (4-hydroxy-2-nonenal) compared to the AL group, suggesting that inhibition of oxidative stress might be a potential mechanism for the neuroprotective effects of EODF
Such complexes have already been thoroughly characterized with regards to impact on redox interaction under environmental stresses
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