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Prodlouen struktura IGF-1LR3 mu umouje vyhnout se vazb na IGF-binding proteins (IGFBPs), kter obvykle omezuj aktivitu pirozenho IGF-1. Tm se zajiuje del psoben a vy biologick innost

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for 6 weeks (form not reported), responded to treatment compared with 69% on anti-depressants alone, a statistically significant difference

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