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Front Neurosci 2018;12:301.ArticlePubMedPMC 67

To shed more light on the identity of the substrate of arGSTs, we performed labelling experiments with H 2 18 O, exploiting the same principle of 18 O incorporation as described earlier

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When SETD1B is functional, mycolactone exposure leads to the upregulation of CHAC1, resulting in decreased GSH levels and increased oxidative stress, ultimately causing apoptosis

doi: 10.1016/j.bodyim.2025.101890

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