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Better et al
Therefore, future research should identify critical regulators that maintain CSC plasticity and tumor heterogeneity
The CYP2E1-induced oxidative stress increases the intracellular GSH level by increasing the activation of GCS through Nrf2 nuclear translocation [74, 75, 84, 17]
Each of the methods has its own advantages and disadvantages
Clin Infect Dis 31:12021208
Moreover, to extend our effort toward the study on the role of canonical autophagy in thyroid carcinogenesis, the conditional autophagyrelated 5 (ATG5)defective ( Atg5 flox/flox ) mice were also employed and used to generate Braf CA/wt , Atg5 flox/flox mice
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