[163, 165] have reported that off-target toxicity in peripheral organs remained minimal, although they emphasised that excessively high doses or positively charged CQDs may still provoke ROS formation, mitochondrial dysfunction, or the activation of inflammatory pathways [163, 165]
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doi: 10.1084/jem.20201707 73 SaltinB.BlomqvistG.MitchellJ
Product Usage: THIS PRODUCT IS INTENDED FOR RESEARCH PURPOSES ONLY
This dual activation mechanism provides redundancy ensuring robust nitric oxide production for vasodilation and vascular protection
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