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Physiologically, this lowering of GSH led to increased LDH release ( P = 0.031), consistent with redox activation of ferroptosis and cell death (Figure 4C and refs

The effects of the active forms of GLP-1 are mediated by a G protein-coupled receptor, GLP-1R, which is expressed in several sites, including enteric and vagal nerves, the stomach, pancreas, intestine, and various brain regions [15]

This dysregulation disrupts normal neural pathways, potentially leading to cognitive dysfunction, particularly in memory encoding and retrieval

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