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In a transgenic mouse model of mutant alpha-synuclein (A53T), experiments showed that alpha-synuclein accumulation in astrocytes affected glutamate transport, triggering a rise of extracellular glutamatergic concentrations and excitotoxicity, which further exacerbated damage to dopaminergic neurons (Gu et al., 2010)

For patients diagnosed with a significant vitamin B12 deficiency, our healthcare team typically begins treatment with more frequent injections, often weekly or even more often, until healthy vitamin B12 levels are restored

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