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Interestingly, defects in mitochondrial energy metabolism are also present in the brains of patients with AD, leading to neuronal apoptosis and A deposition, which increases ROS production by interfering with mitochondrial calcium homeostasis and ETC function

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Preclinical Research Summary Preclinical research on BPC-157 has been conducted primarily in animal models designed to investigate tissue repair and regenerative biological processes

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Does NAC cause cancer