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AD is characterized by a decline in cognitive function in elderly people, accompanied by pathologic features that include -amyloid plaque deposition and neurofibrillary tangles formed by hyperphosphorylated Tau protein (49)

Notably, there is an overlap in cellular and molecular alterations of lamin A protein in both PD and Hutchinson-Gilford Progeria syndrome which develops due to a deficiency of lamin A protein (Al-Kuraishy et al

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