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10.1046/j.1528-1157.44.s10.5.x Epilepsia 45 PelzerN.LouterM

922 In AKT-hyperactivated cancer cells, AKT directly phosphorylates the Ser343 site of TRPML1, inhibits the K552 ubiquitination degradation of TRPML1, and promotes the binding of TRPML1 to ARL8B, leading to lysosomal exocytosis

Funding This work was supported by the Basque Government (IT1620-22)

[DOI] [PubMed] [Google Scholar] 28.Hrelec M., Klicek R., Brcic L., Brcic I., Cvjetko I., Seiwerth S., Sikiric P

The regulatory landscape The FDA has increased scrutiny on peptide compounding