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These findings, although obtained from models of oxidative stress and mitochondrial dysfunction rather than in autism models, are highly relevant because they target biological mechanism that are also characteristic of ASD (Sachdeva et al., 2022)
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Indeed, short-term glutaminolysis induces mTORC1 lysosomal translocation and activation via the Rag GTPase, then inhibiting autophagy and promoting cell growth [88]
Cardone, Platelet Rich Placebo?: Evidence for Platelet Rich Plasma in the Treatment of Tendinopathy and Augmentation of Tendon Repair, Bulletin of the Hospital for Joint Diseases 71 (2013): 57
10.1007/s12013-024-01587-0 88 JeonM.ParkJ.YangE.BaekH
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