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However, whenever the transporter is mutated or deleted, it will disrupt the iron balance and lead to excessive accumulation, triggering cellular oxidative damage and death [72, 73]

Mounting evidence has identified key microorganisms, microbial enzymes, and metabolites as potential therapeutic targets

SLUPP332 is an exercise-mimicking peptide that activates pathways involved in endurance and fat metabolism

A., Ohgami Y., Vega-Villa K

A recent study has shown in vitro that pretreatment with the selective ferroptosis inhibitor, ferrostatin-1, preserved mitochondrial function and mitigated neuronal cell death secondary to isoflurane exposure, suggesting that ferroptosis contributes to isoflurane neurotoxicity [16]

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