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It has been shown that atg5 (an autosomal marker) null mice crossed with Lurcher mice have an increase of PCs death, more than Lurcher mice with atg5, which eventually suggests that atg5 rather protects the PCs in Lurcher mice than causes cell death by autophagy [98]

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These benefits encompass promoting longevity, facilitating wound healing and tissue recovery, aiding DNA repair, and potentially inhibiting tumorigenesis [4, 5, 6, 7, 8, 9]

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