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Consequently, isoacteoside could serve as a potential therapeutic agent for both SAP and SAP-induced AKI

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doi: 10.1002/edm2.432

Therefore, our current study is consistent with a model where, during HFHC dietinduced metabolic dysregulation, the deletion of Dpp4 within Tie2 + cells increases the abundance of GLP-1 delivered to the systemic circulation, enabling the incretin effect, and improves oral glucose tolerance but does not affect HGP