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In a murine myocardial infarction model, miR-92a inhibition improved endothelial cell function and postinfarction recovery by modulating autophagy-related genes in an endothelium-specific manner

PMID: 22783571

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This work was supported by a grant from the Hungarian National Research, Development and Innovation Fund, OTKA K142212 (J.K.), TKP2021-NKTA-21 (J.K.) are gratefully acknowledged

NO also plays a role in the immune response by promoting the activity of macrophages, which are essential for pathogen defence [46]