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Sulforaphane administration in mice exposed to 6-hydroxydopamine has also been shown to improve motor deficits, while dietary intake of sulforaphane preserved dopaminergic neurons following induced dopaminergic neurotoxicity in mice (Morroni et al., 2013

Furthermore, some studies have found that antioxidant therapy may exacerbate disease progression, which may be attributed to the fact that redox processes do not play a singular role in disease development

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Autoimmune encephalitis, especially forms associated with N-methyl-D-aspartate (NMDA) receptor and voltage-gated potassium channel (VGKC) -complex autoantibodies, is characterized by sudden-onset neuropsychiatric symptoms, cognitive dysfunction, seizures, and altered consciousness

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