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Insulin resistance in spontaneously hypertensive rats is associated with endothelial dysfunction characterized by imbalance between NO and ET-1 production

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Furthermore, the effects observed on CREB and BDNF promoter methylation and in regulating miR-34a, miR-212, and miRNA-29c levels, highlighted the role of TIR in overcoming the epigenetic modifications induced by hyperglycemia

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How to cite this article APA-style citation Evenskyn Skin Science Desk, & Kimji, I