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Consistent with other central nervous system injuries, microglia activation in TBI results in different phenotypes, including the pro-inflammatory M1 (neurotoxic) and anti-inflammatory M2 (neuroprotective) phenotypes (Galgano et al., 2017)

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This stress-induced oxidative burden depletes glutathione reserves and may overwhelm cellular antioxidant defenses, potentially contributing to stress-related health consequences

It preps your skin to absorb subsequent treatments more effectively

Growth hormone acutely stimulates skeletal muscle but not whole-body protein synthesis in humans

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