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Description

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Furthermore, aescin substantially reduced cardiac enzymes to levels close to normal, indicating its potential to prevent membrane breakdown and subsequent enzyme leakage 111,112

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A missense mutation (R120G) in B-crystallin leads to abnormal aggregation of both desmin and B-crystallin, resulting in severe cardiomyopathy known as desminopathy

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Since abnormal mitochondrial morphology has been reported as a cuproptosis phenotype in human cultured cells 33,34 , we examined mitochondrial morphology in the pharyngeal muscle of the slcr-46.1 mutant

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72 In contrast, ACSL3 shifts monounsaturated fatty acids (MUFAs) into acyl-CoA esters, which are then integrated into membrane phospholipids, a process that also requires the involvement of LPCAT3, 73 and is closely associated with phospholipid remodeling

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