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These alterations are related to aberrant expression of the bile acid enzyme regulator and bile acid buildup in cells and culture media (Sharanek et al., 2014

These models significantly improve diagnostic accuracy yet still face challenges in interpretability [198]

This imbalance can result from excessive cholesterol secretion by the liver, leading to supersaturation in bile, insufficient bile salts, or lecithin that fails to dissolve cholesterol effectively and impaired gallbladder motility that prevents proper emptying

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this causes dissociation of NADPH oxidase one and nuclear translocation of DPP4, leading to diminished lipid peroxidation and abrogation of ferroptosis-promoting activity (Xie et al., 2017)

Pediatric gastrointestinal bleeding: perspectives from the Italian Society of Pediatric Gastroenterology