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The current model is that most of these compounds induce conformational changes in KEAP1, leading to the inhibition of NRF2 ubiquitylation via dissociation of the inhibitory complex

Protopine protects chondrocytes from undergoing ferroptosis by activating nrf2 pathway

Single-cell and spatial multi-omics analysis reveal that targeting JAG1 in epithelial cells reduces periodontal inflammation and alveolar bone loss

Reversing the inactivation of peroxiredoxins caused by cysteine sulfinic acid formation

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For example, inhibition of the Notch pathway via -secretase inhibitors can lead to compensatory activation of the PI3K/AKT axis, preserving CSC survival