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The main proposed therapeutic mechanisms of AITD include increasing the activity of GPx and TR enzymes in blood plasma and reducing toxic concentrations of H 2 O 2 and lipid hydroperoxides

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Thus, increased ARG availability via CIT or ARG supplementation may not improve endothelial function in individuals with normal FMD [24,61]

Cell 169.1 (2017): 132-147.e16

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Numerous studies, such as genome-wide association studies (GWAS), have identified significance to the expression of numerous genes related to -cell function and insulin secretion