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21 Clinical Presentation Regardless of whether acetaminophen toxicity occurs because of a single overdose or after repeated supratherapeutic ingestion, the progression of acetaminophen poisoning can be described in four sequential phases: preclinical toxic effects (phase one), hepatic injury (phase two), hepatic failure (phase three), and recovery (phase four)

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At 4 weeks of age, GABAA receptor density in the cerebellum was significantly reduced, suggesting that GABAergic inhibitory transmission may be weakened during early developmental stages

Growth Factors 33 , 419429 (2015)

Skin rashes or allergic reactions in rare cases

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