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NO is not only a free radical involved in pathophysiological processes in the oral cavity, as described above

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Mechanistically, the authors reported that the uremic toxin, indoxyl sulfate, activates the aryl hydrocarbon receptor (AhR), and experiments using AhR knockout mice confirmed that AhR activation is a key driver of BBB damage (Bobot et al., 2020)

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In summary, preclinical research consistently demonstrates NACs multifaceted modulation of dopaminergic system, signifying its therapeutic potential across diverse neurological pathologies