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The amount of unattached or "free" unbound bilirubin (Bf) and the level of hydrogen ions (pH) in the circulation determines the likelihood that bilirubin may induce neurological damage

Compared to STZ treatment alone, addition of SFN decreased nephromegaly, glomerular collagen accumulation, glomerulosclerosis, glomerular basement membrane thickness, and urine albumin excretion, but only in wild-type mice [50]

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