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Conclusions from 67 Cu uptake and release measurements performed in hypothalamic slices by the presence of action potential blocker tetrodotoxin [231, 232] or determination of depolarization-induced copper release from nerve endings by AAS [225] raised the concept of copper signalling in the brain

Many of these compounds are prone to degradation in the GI tract because of factors such as varying pH levels, poor water solubility, rapid metabolism and high clearance rates

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The results shown in Fig