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however, in phase 1 clinical trial, BIIB078 failed to exert meaningful effects on CNS pathologies and had no significant impact on neurofilament levels but paradoxically increased inflammatory marker CCL26, and its development has been discontinued (van den Berg et al., 2024

[1] [2] The timing of side effects is closely linked to the drug's pharmacokinetic profile

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A missense variant in mitochondrial amidoxime reducing component 1 gene and protection against liver disease

One primary advantage is the peptides rapid absorption through the nasal mucosa, which the peptide to enter the bloodstream without going through the digestive system which may result in quicker outcomes

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