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doi: 10.1001/jama.2012.164487 66

Retatrutide potently activates the GLP-1R signaling pathway to stimulate glucose-dependent in-sulin secretion through activity at the GIP receptor (GIPR) or the GLP-1R

By preventing the polyubiquitination of phosphorylated IB, the deubiquitinating enzyme CYLD protects GMCs by suppressing the activation of NF-B signaling in a high-glucose environment[44]

is supported by NIH research grants OT2HL161847, OT2OD032581, U24TR001608 and CDC project numbers 75D301-21-P-12254 and 75D301-23-C-18264

Shroff EH, Eberlin LS, Dang VM, Gouw AM, Gabay M, Adam SJ, et al