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[DOI] [PubMed] [Google Scholar] 261.Treitinger A, Spada C, Masokawa IY, et al
Han HS, Choi KY
Several mechanisms have been proposed to explain this: (i) bystander apoptosis occurs due to viral proteins promoting the apoptosis of nearby cells (notably HIV-1 gp120 after its interactions with CD4+ and CCR5 or CXCR4 coreceptors), (ii) immune activation induces FasL production and Fas (CD95) expression in nearby, uninfected CD4+ T cells, resulting in shortened lifespans and an increased sensibility to activation-induced cell death (AICD), and (iii) abortive infection leading to pyroptosis through the caspase-1 pathway, due to an accumulation of incomplete reverse transcripts and the induction of antiviral and inflammatory responses [11]
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8c, more intensive R6G fluorescent signal of 3-PLG-dopa-A-GSH can be seen on the representative images showing brain organoids from healthy or PD organoids compared to the dye of non-targeted 3-PLG-dopa groups (Fig
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