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This potent anti-inflammatory activity is of particular relevance to the treatment of autoimmune thyroiditis (e.g., Hashimotos thyroiditis), where NF-B-mediated inflammation is a key driver of thyroid follicular cell destruction(Zheng et al., 2022)

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An important part of this is that FOXO4 lives in the nucleus of the cell and p53 does its work at the mitochondria two different parts of the cell

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More specifically, the researchers posited that the peptide may have enhanced the expression of Ki-67 and CD98hc that are less intensively synthesized during cell aging