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The bidirectional interplay of -synuclein with lipids in the central nervous system and its implications for the pathogenesis of Parkinsons Disease

Together, our data suggest that p53 controls a stress-responsive gene programme, rather than a single downstream gene, that facilitates fasting-dependent recruitment of macrophages into eWAT under metabolic stress

Regulating blood pressure through hormone secretion

Notoginsenoside R1 attenuates ischemic heart failure by modulating MDM2/ arrestin2-mediated 2-adrenergic receptor ubiquitination

A series of treatments is recommended for optimal resultsusually once or twice a week

Annu Rev Neurosci