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In addition to L-cellspecific Bmal1 deletion, which directly establishes the requirement of BMAL1 for rhythmic GLP-1 release, mice lacking other core clock components (including Clock and Period genes) exhibit disrupted feeding behavior, impaired glucose homeostasis, and abnormal daily patterns of incretin secretion [13,19,50,51]

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Post-meal glucose: The combination of enhanced insulin release, suppressed glucagon, and slowed gastric emptying substantially reduces glucose spikes after eating

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