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Inflammation and Cytokine Regulation 4.1.1

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Of note, the exaggerated levels of pro-inflammatory mediators and the suppressed AKT level are considered the key contributors in disrupting BDNF signaling, which lead to consequent enhancement in neuronal apoptosis and impairment in memory functions (Rossi et al

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Moreover, the acute lipid-lowering actions of GLP-1RA are independent of the control of islet hormone secretion or gastric emptying and inhibited by the GLP-1R antagonist exendin (939), consistent with a key role for the canonical GLP-1R in the control of postprandial TG secretion (9, 10)

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