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Many studies have investigated the potential correlation between genetic background and hypogonadal dysfunction in individuals with PWS, but no data have shown an impact of the various genetic defects (such as 15q11-13 paternal deletion, mUPD, or imprinting defect) on pubertal development and fertility (203, 204)
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Of course, some couples may be open to that delay if they are actively pursuing metabolic health support for the woman
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