For hip injuries including bursitis, tendinopathy, and soft tissue damage, typical protocols involve 0.25 to 0.5 mg daily via subcutaneous injection near the affected area for 4 to 8 weeks
Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
shunting of glucose into the PPP instead of into pyruvate generation
The liver and mitochondria require adequate antioxidants, minerals, and amino acids to neutralize oxidative byproducts generated by medications, inflammation, and environmental exposures
Vitamin C also participates in redox recycling of other important antioxidants
The increased local blood supply brings to the area nutrients and cells (fibroblasts, osteoblasts and chondroblasts) that facilitate the repair process