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(PubMed) Joseph MA, Moysich KB, Freudenheim JL, et al

However, SGLT2-i could reduce renal and extra-renal glucotoxicity by inhibiting many pro-inflammatory pathways such as low serum ketone levels, hyperuricemia, oxidative stress and advanced glycation-end products (AGEs) induced by hyperglycemia ( Indeed, there is a growing body of evidence suggesting a significant beneficial class effect associated with SGLT2 inhibition, resulting in MACE, cardiovascular death and hospitalization for heart failure (HHF) reduction, despite some variabilities in the findings provided by individual CVOTs (40)

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The histogram shows percentages of red cells (non-replicators) and white cells (non-red, replicators) in a population, determined by a flow cytometers red-fluorescence detector that quantified redness of individual cells

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